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Kimford J. Meador, M.D uk pharmacy ., Gus A. Baker, Ph.D., Nancy Browning, Ph.D., Clayton-Smith Jill, M.D., Deborah T. Combs-Cantrell, M.D., Morris Cohen, Ed.D., Laura A. Kalayjian, M.D., Andres Kanner, M.D., Joyce D. Liporace, M.D., Web page B. Pennell, M.D., Michael Privitera, M.D., and David W. Loring, Ph.D. For the NEAD Study Group: Cognitive Function at three years old after Fetal Exposure to Antiepileptic Drugs Women with epilepsy are in increased risk for poor pregnancy outcomes, although the majority of their kids are normal.1 In animals, fetal contact with antiepileptic drugs at doses lower than those that result in structural malformations can produce behavioral and cognitive deficits, alter neurochemistry, and reduce mind weight.2,3 The effects of in utero exposure to antiepileptic drugs in individuals may contribute to poor neurodevelopmental outcomes, 4 but this risk should be balanced against potentially grave risks to both mother and fetus posed by seizures.

This rate of repeat revascularization is lower than the rates reported in prior comparative trials involving patients with less-complex clinical profiles and lesions.10 The increase in the rate of repeat revascularization with PCI as compared with CABG did not appear to translate into a significant overall increase in the death rate or myocardial infarction, although longer-term follow-up is needed. The risk of repeat revascularization after PCI needs to be balanced against the invasiveness of CABG and the chance of stroke, as previously reported in a meta-analysis of 23 research evaluating CABG and PCI, in which procedure-related strokes were found to become more common after CABG , without a concomitant decrease in survival.34 Recently, concern has been expressed about the chance of an increased risk of past due stent thrombosis with drug-eluting stents.